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Additionally, SUMOylation of transcription factors impairs their nuclear localization, further disrupting hormonal signaling cascades (62, 63)

To overcome this issue, some engineering processes envisage the loading of said binding proteins in order to increase the binding capacity of RBCs

By preserving NAD+ and SAM in both fat and muscle tissue, NNMT inhibition may theoretically address the metabolic dysfunction underlying both excessive fat storage and impaired muscle maintenance