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The discovery of SQORs unique cysteine trisulfide bond cofactor explains its remarkable catalytic efficiency ( 2 S levels, keeping them within an optimal physiological range: on the one hand, genetic defects (e.g., in Leigh syndrome) or CoQ deficiency that abolish SQOR activity can cause toxic H 2 S accumulation, inhibiting the mitochondrial respiratory chain and leading to severe energy failure

The activated p53 translocates and accumulates in the nucleus, which then binds to and activates the EBV Rp

FEBS Open Bio 16, 382396

Regulation of JNK signalling by GSTp

Antiretrovirals may also cause damage to cells in a person with HIV, decreasing function of crucial cells and releasing toxins in the body