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Mutations in neuronal synaptic proteins include post-synaptic proteins such as neuroligins (NLGNs) and pre-synaptic proteins such as neurexins (NRXN), SHANK, and GRIP1 [73]

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The balance between these opposing outcomes is governed by ectonucleotidases, which control the conversion of ATP to adenosine [326]

Moreover, the systemic elevation of Gln improves insulin sensitivity and restores glucose homeostasis in mouse models of obesity