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Description

The pathogenesis of POAG can be summarized as follows: endoplasmic reticulum (ER) stress promotes the excessive deposition of ECM in the trabecular meshwork (TM) by interfering with ECM protein processing, thereby inducing TM fibrosis

BPC-157 sits in the pre-clinical stage for reproductive health, though its anti-inflammatory and gut-protective effects are relevant to the PMOS disease profile