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Emerging evidence reveals that the amino acid oxidase interleukin (IL)-4-induced-1 (IL4i1) can produce indole-3-pyruvate (In3Py) to inhibit ferroptosis through radical scavenging and altering a gene expression profile (44) ( Figure 1 )

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Additionally, a previous paper indicated that NINJ1 deletion in mouse bone marrow-derived macrophages (BMDMs) or MEFs conferred prolonged protection against cell lysis, as measured by LDH release, but it did not prevent cell death, as determined by ATP production