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coli Based on our findings that the glutathione redox homeostasis can be fully restored by the addition of endogenous reduced glutathione to a gshA mutant lacking the transporter Gsi, essentially with the same velocity as in a mutant lacking only GshA, we hypothesized the presence of a yet unidentified glutathione transporter specific for reduced glutathione

These physiological abnormalities as well as studies addressing treatments of these abnormalities are reviewed in this article

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Nutrients 13 (6), 1898

With increase in toxin exposure, the demand for glutathione production also rises