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Extracellular vesicles (EVs)-encompassing exosomes, microvesicles, retrovirus-like particles, and apoptotic bodies-serve as key intercellular mediators that inhibit ferroptosis through activation of the Nrf2 pathway and subsequent upregulation of the antioxidant gene SLC7A11 (90)

However, studies have yet to recapitulate the symptoms of ASD, which remains a highly laudable goal of research in the field

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The chemical diversity of the bioactive compounds produced by these bacteria is considerably more than what has been exploited to date [63, 64]