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Additionally, the BMSC/SA/CoI/SDF-1 scaffold could facilitate BMSC migration in lesions and partially boost neurogenesis by triggering the FAK/PI3K/AKT pathway (Wang et al., 2020a), which was regulated by SDF-1/CXCR4 and could contribute to the reduction in brain damage, neuronal death, and neural inflammation (Ma et al., 2021)

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In both cases, oxidative stress is thought to be the common underlying mechanism that leads to cellular dysfunction and, eventual cell death