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To validate the phosphorylation relationship between PEPR1 and BAK1 in vivo , we co-expressed PEPR1-Km-GFP with BAK1-mCherry in the Nicotiana benthamiana leaf epidermis with or without Pep1 (500 nM) treatment for 15 min, and performed MS-based phospho-analysis (Supplemental Dataset 2)

During the pathogenesis of OA, JUN directly binds to the promoter of NCOA4 and initializes its transcription, increasing ferritinophagy degradation, leading to ferroptosis and extracellular matrix degradation in chondrocytes

[DOI] [PMC free article] [PubMed] [Google Scholar] 346.Wu Y, et al

The patch delivers reliable support for antioxidant defence, detoxification, and immune health throughout the day