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Role of G6PD-deficiency-mediated RBC hemolysis in ASCVD Any discussion of the potential impact of G6PD-d on ASCVD would be incomplete without considering how the predominant hemolytic phenotype may contribute to worsened disease

Caspase proteolysis of desmin produces a dominant-negative inhibitor of intermediate filaments and promotes apoptosis

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The intracellular GSH is decreased with the increased CCF concentration, accompanied by increased tumor cell apoptosis (Figure 5B), which demonstrates the antitumor effect of GSH-promoted CDT

Networking antioxidants in the isolated rat heart are selectively depleted by ischemia-reperfusion