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reported an increase in the levels of dysfunctional mitochondria and mitochondrial membrane proteins, translocase of the outer mitochondrial membrane 20 (Tom20), translocase of the inner mitochondrial membrane 23 (Tim23), and porin in the ASD temporal cortex without a change in the transcription of genes, such as PARK2 , responsible for the activation of mitophagy

The multifaceted role of mitochondria in ASD is schematically presented in Fig

Enhanced lipid utilization in infants receiving oral L-carnitine during long-term parenteral nutrition

Inter-organ cross-talk in metabolic syndrome