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Here, we demonstrate that FHOD3L nucleates actin and rapidly but briefly elongates filaments after temporarily pausing elongation, in vitro

Together, this data argues that in mammalian cells, the Sec61 translocon does not participate in the Ca 2+ depletion- or CsA-induced redox shift apart from serving as a Ca 2+ leak channel in the presence of puromycin

Indeed, TNF- has been shown to modulate many dysfunctional processes in BD including synaptic plasticity, neurotransmission, neurogenesis, neuronal survival, apoptosis, and even cognitive/behavioral functioning [4, 5, 17]

A brain-based pain facilitation mechanism contributes to painful diabetic polyneuropathy

doi: 10.1038/s41423-022-00859-0 179 ChenNXieQ-MSongS-MGuoS-NFangYFeiG-Het al