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Scagliotti, J.L

pmid:9888458 32

Defects in cellular ATP production found in several regions of PD brains have been associated with mitochondrial dysfunction 71 , and previous studies showing that HPRT1 deficiency inhibits mitochondrial protein complex I-dependent respiration 72 indicate that HPRT1 may be involved in energy metabolism dysregulations in PD

In T-cell lymphomas, TP53 mutations can lead to impaired apoptosis and increased survival of malignant cells despite radiation-induced DNA damage, suggesting a potential for reduced radiation sensitivity (154)