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DNA damage activates PARPs that can use up to 90% of the cell's NAD+, which makes DNA repair heavily reliant on NAD+ availability

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Similarly, a roGFP2-iL-Grx1 fusion redox probe targeted to the ER of Arabidopsis thaliana cells lacking Ero1/2, the functional homolog of DsbB in eukaryotes was more reduced, and re-oxidation after a reductive pulse was inhibited by the lack of Ero1/2 (Ugalde et al., 2022)

[DOI] [PubMed] [Google Scholar] 192.DAmico F, Danese S, Peyrin-Biroulet L

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