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doi:10.3390/ijerph15061188 159

Homocysteine may also react with nitric oxide in the body to form S-nitrosohomocysteine, a compound that causes endothelial damage and reduces the vasodilatory function of blood vessels.2,21 The primary goal in treating hyperhomocysteinemia is to reduce elevated plasma homocysteine levels in order to minimize the risk of neurological and vascular complications