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It influences satellite cell activation and promotes intracellular pathways associated with protein synthesis and tissue regeneration

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Randomized Cross-over Trial of Short-Term Water-Only Fasting: Metabolic and Cardiovascular Consequences

In endothelial cells (ECs), copper overload disrupts mitochondrial dynamics, increases fission and ROS production, and ultimately leads to cell death [34] In cardiomyocytes, copper promotes hypertrophy and inflammation [35], disrupts fatty acid and lipid metabolism, upregulates autophagy-associated proteins and genes, and impedes calcium ion uptake by the sarcoplasmic reticulum, thereby inhibiting myocardial contractility [36]

In a rat model of HFD-induced MASLD, serum levels of IL-22 were markedly reduced, while the hepatic expression of the autophagy marker LC3B was increased