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NIK activation by TNFRs, such as CD40 and BAFF receptor (BAFFR), involves degradation of TRAF3, which causes liberation and stabilization of NIK, triggering the activation of downstream non-canonical NF-B pathway (Fig

44 Subsequent work has definitively demonstrated a crucial role for c-Rel and RelA in mediating induction of the Th17 lineage transcription factor RORt and the generation of Th17 cells

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Updated review of mechanisms

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