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PHDs can be pharmacologically inhibited by a new class of drugs termed prolyl hydroxylase inhibitors which have recently been approved for the treatment of anemia associated with chronic kidney disease

[11] Dies war historisch ein Einwand gegen die orale Glutathion-Supplementierung

Chun, K., Raut, P

The rax1-1 mutant is characterized by an enhanced APX2 gene expression due to a lesion in the gene encoding GSH1 concomitantly with a 50% reduced GSH content