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Similarly, mitophagy enhancers (e.g., urolithin A) and metabolic regulators (e.g., nicotinamide riboside, metformin) have shown promising activity in cellular and animal models, but their therapeutic effects in human PD (mitochondrial dysfunction evolving over decades) are largely unknown [135,136,137,138]

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F2,6BP is structurally similar to fructose 1,6-bisphosphate but is not an intermediate in either glycolysis or gluconeogenesis